A further issue of probable problem will be the possible neuroinflammatory outcomes of psychostimulants. In rats, MPH administration has become noted to generate neuroinflammation and oxidative pressure in the hippocampus and cerebral cortex, as measured through the inflammatory markers tumor necrosis element α and interleukin 1β (264, 265). In a
• Intracortical MPH amplified cortical cell excitability in rats, an outcome that was lost subsequent catecholamine depletion and was mediated by means of stimulation of αtwo adrenergic receptors although not D1 receptors*Publications have been excluded if they did not focus on the system of action or pharmacologic consequences of amphetamine or
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